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China β-Nicotinamide Mononucleotide Raw Material β-Nicotinamide Mononucleotide Powder Factory Supply β-Nicotinamide Mononucleotide Raw Material, High Quality NMN Powder Purity 99% Nutritional supplements NMN Powder, Also NMN Powder is used in anti-aging research.
Manufactory Supply β-Nicotinamide Mononucleotide Raw Material Powder, β-Nicotinamide Mononucleotide Powder and Pharmaceutical β-Nicotinamide Mononucleotide High Quality NMN Powder.
Uses and functions of β-Nicotinamide Mononucleotide Raw Powder
In mammals, β-nicotinamide mononucleotide powder is produced from nicotinamide (Nam) catalysed by Nampt (a protease in vivo), and subsequently nicotinamide mononucleotide is catalyzed by nicotinamide mononucleotide adenylyltransferase to produce NAD+.
Nicotinamide mononucleotide is a direct way to replenish NAD+. It is by significantly enhancing the ability to repair intracellular DNA damage that nicotinamide mononucleotide ultimately reverses the effects of aging.
Nicotinamide mononucleotide is converted into nicotinamide adenine dinucleotide (NAD), which is essential for energy metabolism in the body. In mouse tests, it has been demonstrated that nicotinamide mononucleotide activates a gene called acetylase in the body, which has been shown to be effective in prolonging life and treating diabetes.
NAD is a substance that can be produced by the human body, and studies have shown that the amount of NAD in the body decreases with age.
Pharmacological Effects of NMN Raw Powder
Nicotinamide mononucleotide, also known as β-nicotinamide mononucleotide (NMN), is a synthetic substrate for coenzyme I. Meanwhile, NMN has also been used in anti-aging research.
Studies have shown that β-NMN can also play a regulatory role in insulin secretion and has an effect on the expression level of mRNA. β-NMN has a wide range of applications in the field of medical therapy.
β-Nicotinamide mononucleotide is a product of the extracellular nicotinamide phosphoribosyltransferase (eNAMPT) reaction and a key NAD+ intermediate.
It improves glucose tolerance by restoring NAD+ levels in HFD-induced T2D mice. It also enhances hepatic insulin sensitivity and restores gene expression associated with oxidative stress, inflammatory responses and circadian rhythms partly through SIRT1 activation.
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