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  • HRK Factory Palmitamide Mea Raw Materials Powder on sale

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    Product Overview:

    Hexadecanamide ethanol (PEA) is an endogenous fatty acid amide that belongs to the class of nuclear factor agonists. It has been shown to bind to receptors in the nucleus of cells (nuclear receptors) and exert a large variety of biological functions related to chronic pain and inflammation.

    HRK Factory Palmitamide Mea Raw Materials Powder on sale Attributes

    Palmitamide Mea Raw Materials Powder

    CAS:544-31-0

    MF:C18H37NO2

    Palmitamide Mea

    MW:299.49

    EINECS:208-867-9

    Specification​: 99% min Palmitamide Mea Powder

    Sample:Palmitamide Mea Powder

    Packaging:1kg/bag, 25kg/drum

    Brand: Raw Materials

    Appearance:white

    Storage: Cool Dry Place

    Shelf Life: 2 Years

    Test Method: HPLC

    HRK Factory Palmitamide Mea Raw Materials Powder on sale Details

    Palmitamide Mea Usage and Function.

    Hexadecanamide ethanol is an organic synthesis intermediate and pharmaceutical intermediate that can be used in laboratory R&D processes and in chemical and pharmaceutical R&D processes.

    Palmitamide Mea Powder

    Uses of Palmitamide Mea.

    The main uses of cetamidoethanol include anti-inflammatory and analgesic, enhancement of immunomodulation, treatment of chronic pain, epilepsy, cerebral ischaemia and stroke, Alzheimer's disease and Parkinson's disease. It is also widely used, particularly in sports nutritional supplements and allied health formulations, and these uses reflect the important role of cetamidoethanol in healthcare, especially in the field of naturopathy, where it offers a better alternative as a natural painkiller compared to the possible side effects of conventional medications.

    Palmitamide Mea

    In vitro studies of Palmitamide Mea.

    PEA effectively prevented glutamate toxicity in murine cerebellar granule cells and enhanced microglia motility. In mitochondrial fractions from cells stimulated with PEA, the expression of steroid synthesis-rapid regulator protein (StAR) and P450scc, which include some neurosteroid-forming proteins, was increased.PEA also had protective effects: e.g., reduction of malondialdehyde formation in cells treated with L-buthionine-(S,R)-sulfoximine, which is the cause of glutathione deficiency.The effect of PEA was partially mediated by finasteride (5a-reductase inhibitor). The effect of PEA can be partially inhibited by finasteride (5a-reductase inhibitor).

    Preparation of Bulk Palmitamide Mea Powder.

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