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Polydatin Raw Materials Powder
Product Overview:
It is the root of Polygonum cuspidatum Sieb. et Zucc. and the root of Polygonum cuspidatum. Mainly from Polygonum cuspidatum Sieb. et Zucc. Polygonum cuspidatum dry rhizome and root. Tiger Balm glycoside is one of the natural active ingredients.
Polydatin Raw Materials Powder Attributes
CAS:65914-17-2
MF:C20H22O8
MW:390.38
EINECS:200-001-8
Specification: 99% min Polydatin Powder
Sample: Polydatin Powder
Packaging:1kg/bag, 25kg/drum
Brand: Henrikang
Appearance: White Powder
Storage: Cool Dry Place
Shelf Life: 2 Years
Test Method: HPLC
Polydatin Raw Materials Powder Details
Polydatin Powder Usage and Synthesis.
Polydatin is the root of Polygonum cuspidatum Sieb. et Zucc. and the root of Polygonum cuspidatum. Mainly from Polygonum cuspidatum Sieb. et Zucc. Polygonum cuspidatum dry rhizome and root. Tiger Balm glycoside is one of the natural active ingredients.
Uses and functions of Polydatin.
Anti-cough, anti-phlegm, asthma, antibacterial, scavenging free radicals, regulating blood lipids, lowering cholesterol. Antibacterial effect:Inhibit Staphylococcus aureus, Cattacoccus, Streptococcus A or B, Escherichia coli and so on.
Pharmacological Effect of Polydatin.
Polydatin improves microcirculation, inhibits the release of lysosomal enzymes, antioxidant, prevents leukocyte sticking in microcirculation, hypolipidemic, anti-lipid peroxidation, antipathogenic microorganisms, antitussive, asthmatic, antibacterial, antiviral, hepatoprotective and other effects. The drug thujaplicin injection can treat myocardial ischemia, cerebral ischemia, shock and other cardiovascular and cerebrovascular diseases.
In vivo studies of Bulk Polydatin Powder.
In cecum ligation perforation (CLP-)-induced sepsis mice, pretreatment with polydatin (15, 45, and 100 mg/kg) dose-dependently reduces sepsis-induced mortality and lung injury by inhibiting CLP-induced production of serum tumour necrosis factor-alpha (TNF-alpha) and interleukin-6 (IL-6), the protein expression of lung cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase isoforms (iNOS), and the activation of NF-κB