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  • dexibuprofen Raw Materials Powder

    • dexibuprofen Raw Materials Powder
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    Product Overview:

    Dexibuprofen is the dextrose form of ibuprofen. It has been found that the pharmacological activity of ibuprofen is mainly derived from the dextrose form, and that it has a higher efficacy than the equivalent dose of ibuprofen, with therapeutic effects being achieved with smaller doses.

    dexibuprofen Raw Materials Powder Attributes

    dexibuprofen Raw Materials Powder

    CAS:51146-56-6

    MF: C13H18O2

    dexibuprofen Powder

    MW: 206.28

    EINECS: 610-620-9

    Specification​: 99% min dexibuprofen Powder

    Sample: dexibuprofen Powder

    Packaging:1kg/bag, 25kg/drum

    Brand: Henrikang

    Appearance: White Powder

    Storage: Cool Dry Place

    Shelf Life: 2 Years

    Test Method: HPLC

    dexibuprofen Raw Materials Powder Details

    dexibuprofen Raw Powder Usage and Synthesis.

    There is an isomerase (2-arylpropionyl-CoA epimerase) in the body that converts (R)-isobutylpropionic acid to (S)-(+)-isobutylpropionic acid. Because of this, and because of the high cost of synthesising compounds with special optical selectivity, only racemic products are currently on the market. Dextro Ibuprofen is the right-handed form of Ibuprofen, a non-steroidal antipyretic, analgesic, and anti-inflammatory drug, and the right-handed form is more potent than the racemic and levitra forms. In the body, 60% of the levosome, can be converted to the dextrosome, for for the overall level of the drug to provide an active reserve.

    dexibuprofen Raw Materials

    Uses and functions of dexibuprofen Powder.

    Antipyretic and analgesic anti-inflammatory drugs.

    dexibuprofen Powder

    Pharmacological dexibuprofen Raw Materials Powder.

    (S)-(+)-Ibuprofen(HCT-15andHCA-7cells;0-1000µM;8days)treatmentreducesconcentrationdependentlycellsurvivalinbothcelllinestoasimilarextent.(S)-(+)-Ibuprofen(HCT-15andHCA-7cells;0-1000µM;20-72hours)treatmentcausesaG0/G1phaseblockaswellasapoptosis.(S)-(+)-Ibuprofen(HCT-15andHCA-7cells;9Chemicalbook00µM;4-72hours)treatmentshowsadownregulationofcyclinAandBandanincreaseofthecellcycleinhibitoryproteinp27Kip-1.(S)-(+)-IbuprofeninhibitsCOXactivity,thromboxaneformation,andplateletaggregation.(S)-(+)-IbuprofeninhibitstheactivationofNF-κBinresponsetoT-cellstimulationwithanIC50of61.7μM.

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